Marijuana is a more appropriately named cannabis sativa herb. Some cannabis sativa plants, as described, do not have abuse potential and are referred to as hemp. Hemp is used extensively for various fibre items like newspaper and artist’s canvas. What we call marijuana is Cannabis sativa with violence potential. It is important to note that while studies have been popular for many years, there is a great deal that researchers still do not know about marijuana. The effects of marijuana are known to neuroscientists and biologists, but they still do not completely understand why.Have a look at Flow with the high of recreational marijuana from Cannasseur Pueblo West for more info on this.
THC specifically impacts the central nervous system (CNS) as a psychoactive substance. A wide variety of neurotransmitters are affected and other biochemical and enzymatic activity is also catalysed. When the THC stimulates various neuroreceptors in the brain, the CNS is activated, allowing the different physical and emotional responses to be more specifically described. Substances that imitate chemicals that the brain produces naturally are the only substances that can cause neurotransmitters. The fact that THC activates the activity of the brain teaches scientists that the brain has natural receptors for cannabinoids. Why humans have natural cannabinoid receptors and how they function is still unknown. What we do know is that cannabinoid receptors are activated by marijuana up to twenty times more actively than any of the body’s normal neurotransmitters will ever be.
The relationship between THC and the neurotransmitter serotonin is probably the biggest mystery of all. All psychoactive drugs, but more importantly alcohol and nicotine, are among the most activated by serotonin receptors. Serotonin is still a little known neurochemical independent of the relationship of marijuana with the chemical, and its supposed neuroscientific working and intent functions are still largely conceptual. What neuroscientists have definitively discovered is that there are very high levels of serotonin production among marijuana smokers. I would hypothesise that this relationship between THC and serotonin may explain the “marijuana maintenance programme to achieve alcohol abstinence and enable marijuana smokers to avoid painful symptoms of withdrawal and avoid alcohol cravings. The usefulness of “marijuana maintenance” is not empirical in helping alcohol abstinence, but it is a phenomenon I have personally experienced with multiple clients.
Interestingly, marijuana mimics so many other drugs’ physiological responses that it is highly difficult to group it into a particular class. It can be put in each of these groups by researchers: psychedelic; hallucinogen; or serotonin inhibitor. It has properties that mimic opioids with similar chemical responses. Stimulants are imitated by other chemical responses.Classifies marijuana – cannabinoids – into its own separate class. The explanation for this misunderstanding is the ambiguity, both known and unknown, of the various psychoactive properties found within marijuana. One recent client I saw could not recover as long as he was still smoking marijuana from the visual distortions he experienced as a result of pervasive psychedelic use. This was apparently due to the psychedelic qualities present in active cannabis. Marijuana was strong enough to keep the brain from healing and recovering, but not strong enough to create these visual distortions on its own.